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Reading
is the most important skill that a child must acquire at school, because one
must learn to read to be able to read to learn. The implication of this is that
the child who is a poor reader will usually also be a poor learner.
Unfortunately poor reading skills, and therefore poor learning skills, have
become a reality for an alarming number of people. The $14 million National
Adult Literacy Survey of 1993 found that even though most adults in this survey
had finished high school, 96% of them could not read, write, and figure well
enough to go to college. Even more to the point, 25% were plainly unable to
read.
Even more
alarming is that reading difficulties are not limited to people who are
environmentally, culturally or economically disadvantaged. Many children come
from good homes, go to good schools and score average to above average on IQ
tests. Yet, they battle to learn to read, and many never succeed.
Children
with reading difficulties share a number of common symptoms. They are inclined
to reverse letters or words, to omit letters, to lose their place, to remember
little of what they have read, or to read with poor comprehension. These
children are considered to suffer from a learning disability (LD), commonly
called dyslexia.
According
to the Orton Dyslexia Society at least one in every ten of otherwise able people
has serious dyslexia problems. The Foundation of Children with Learning
Disabilities states that learning-disabled children represent more than ten
million of the total population of the U.S.A. Estimates of learning-disabled
students being dyslexic vary between 70 and 80 percent.
Find The Cause to Find
A Cure
Most
problems can only be solved if one knows what causes the problem. A disease such
as scurvy claimed the lives of thousands of seamen during long sea voyages. The
disease was cured fairly quickly once the cause was discovered, viz. a Vitamin C
deficiency. A viable point of departure in LD research would therefore be to ask
the question, "What is the CAUSE of dyslexia?"
The idea
that dyslexia is a certifiable biological disorder, a physical problem that
could be diagnosed and treated accordingly, gained credence during the 1960s and
1970s, giving rise to an armada of theories. One such a theory states that
dyslexia is the result when the link between the language, hearing and
comprehension centers of the brain is somehow misconfigured during fetal
development. Another theory states that dyslexia is caused by "faulty wiring in
the brain," whereas another holds that a subtle impairment of vision may be
responsible, while yet another believes that a cerebellar-vestibular dysfunction
may be responsible for the learning disability. All these theories – most of
them blaming some difference in structure between the brain of the dyslexic and
that of the so-called normal reader – have lead to nothing at all. Despite all
these theories and all the intervention efforts based on them, not to mention
the vast amounts of money expended in the process, the numbers of dyslexics
continue to escalate.
Except
for the fact that proof of a neurological deficit still eludes the researchers,
this theory leaves many questions unanswered. If dyslexia has a neurological
basis, why is this supposedly non-contagious "ailment" on the increase? Compare
the present situation with, for example, that of a century ago. In 1910 the
literacy rate in the U.S.A. was so high it was predicted, "the public schools
will in a short time practically eliminate illiteracy." In 1935, a survey of the
375,000 men working in the Civilian Conservation Corps – a government-sponsored
work project to provide employment – found an illiteracy rate of 1.9 percent. It
is most noteworthy that this last figure was found among men primarily of low
socio-economic status. It is even more noteworthy that the illiteracy rates of
the first half of the twentieth century reflected, for the most part, people who
had never had the advantage of schooling.
It is
also impossible to explain how a neurological dysfunction can be more prevalent
in specific areas or countries. While the National Commission on Excellence in
1983 warned that the American nation was "at risk," remedial reading facilities
were not needed at all in Japan due to the rarity of reading problems. Some
would argue that reading problems were virtually nonexistent in Japan because
their written language is easier than our Latin alphabet. That, however, is
simply not true. The Japanese Kanji ideograms consist of 1,850 characters. In
addition there are two Kana syllabaries, which – like our Latin alphabet – use
symbols to represent sounds. Each Kana syllabary has 46 basic letters compared
to our 26.
Differences in Brain Structure Not the
Equivalent of a Neurological Disorder
It is
also important to note that differences in brain structures do not necessarily
equal brain disorders. Differences between dyslexics’ brains and those of normal
readers are not necessarily the cause of a reading difficulty. Such differences
can well be the EFFECT of a learning difficulty.
Latest
neurological findings – for example through the work of Michael Merzenich of the
University of San Francisco – show that, while certain areas of the brain are
designated for specific purposes, brain cells and cortical maps do change in
response to learning. An interesting study in London has found that an area of
the brain associated with navigation was larger in London’s famed taxi drivers
than in other people. The drivers’ brains have adapted to help them store a
detailed mental map of the city, shrinking in one area to allow growth in
another.
The
tendency over the past nearly a century has been to try and fit the dyslexia
shoe on the foot of the children who fail to learn to read. All efforts to make
this shoe fit have failed. If a shoe does not fit one foot, shouldn’t we try it
on the other foot?
Putting the Shoe on the Other Foot
When a
person fails to learn something, there are at least two possible reasons why he
failed. The first is that there may be something wrong with the person. The
second is that there may be something wrong with the way in which he was taught.
Unfortunately most people have so far jumped to the very hasty conclusion that,
when the otherwise normal child fails to learn to read, it must be the first
possibility that applies.
When
teaching, it is imperative to take note of the fact that learning is a
stratified process. One step needs to be mastered well enough before subsequent
steps can be learned. This means that there is a sequence involved in learning.
It is like climbing a ladder; if you miss one of the rungs of the ladder, you
will fall off. If you miss out on one of the important steps in the learning
process, you will not be able to master subsequent steps.
A simple
and practical example of this is the fact that one has to learn to count before
it becomes possible to learn to add and subtract. If one tried to teach a child
to add and subtract before he had been taught to count, one would quickly
discover that no amount of effort would ever succeed in teaching the child these
skills. Conceivably people who abide by the learning disabilities idea would
then conclude that the child suffered from a neurological dysfunction, or from
"dyscalculia," overlooking that the ability to count must be acquired FIRST,
BEFORE it becomes possible to learn to add and subtract.
This
principle is also of great importance on the sports field. If we go to a soccer
field to watch a soccer coach at work, we shall soon find that he spends much
time drilling his players on basic skills, like heading, passing, dribbling,
kicking, etc. The players who are most proficient at these basic skills usually
turn out to be the best players in the actual game situation.
In the
same way, there are also certain skills and knowledge that a child must acquire
FIRST, BEFORE it becomes possible for him to become a good reader. Basic skills
like concentration, visual discrimination, accurate perception and memorizing,
skills of association, auditory memory and lateral interpretation are all
functions that form the foundation of good reading and spelling. Until a child
has mastered these basic skills first, reading will remain a closed – or at most
half-open – book to him.
Teaching
these basic skills used to form part of the educational system for many
centuries, but have since been removed from Western education by "innovative"
educators such as John Dewey and his cohorts. In this way the epidemic that is
now called "dyslexia" was created.
Already
in 1974, in "Reading Teacher," Bateman suggested that the term "learning
disabilities" be replaced by "teaching disabilities." The focus, he said, should
be on the inadequate skills of the adults who are supposed to teach the
children, instead of on blaming the children of mysterious brain dysfunctions.
In 1987 Dr. Thomas Armstrong coined the word "dysteachia" to refer to children
suffering from "pedagogical illness" or inappropriate teaching strategies.
Perhaps
it is time that we investigate the possibility that Bateman and Armstrong may
have been correct when they said that the shoe was on the wrong foot.
Author Bio: Susan du Plessis is the co-author of "The Right to Read;
Beating Dyslexia and Other Learning Disabilities," and the author or co-author
of four other books on learning and learning disabilities. She has been involved
in helping children reach their full potential for 15 years. She holds BD and BA
Hons (psychology). Visit her website at
http://www.audiblox2000.com
Other articles by Susan du Plessis:
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Other Helpful
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NOTE: The article above titled "Dyslexia:
Is the Shoe Perhaps on the Wrong Foot?" was provided by a
visitor to "The New Parents Guide" and is the opinion of its author
Susan du Plessis. "The New Parents Guide" does not guarantee the information to
be factual. Always use the guidance of your child's doctor
over information you read on this site or elsewhere; your doctor knows what is
best for your baby.
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